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Raw Semax powder (80714-61-0)

Raw Semax powder regulates the expression and the activation of the hippocampal (brain-derived neurotrophic factor) BDNF/(tropomyosin………


Status: In Mass Production
Synthesized and Customized Available
Capacity: 1277kg/month

Description

Raw Semax powder (80714-61-0) video

 

Raw Semax powder (80714-61-0) Description

Raw Semax powder regulates the expression and the activation of the hippocampal (brain-derived neurotrophic factor) BDNF/(tropomyosin receptor kinase B) trkB system. It enhances selective attention at the time of information reception, increases memory consolidation and stimulates learning abilities. Raw Semax powder is an active element of Raw Semax powder drugs, which is used for the treatment of brain hypoxia and ischemia. It stimulates the activity of choline acetyltransferase. Raw Semax powder exhibits properties of the melanocortin (MC) receptor antagonist.

Raw Semax powder (80714-61-0) Specifications

Product Name Raw Semax powder
Chemical Name Pro-gly-pro-acth (4-7); ACTH (4-7), Pro-Gly-Pro-; ACTH (4-7), prolyl-glycyl-proline-; L-Proline, 1-(N-(1-(N-(N-(N-L-methionyl-L-alpha-glutamyl)-L-histidyl)-L-phenylalanyl)-L-prolyl)glycyl)-
Sequence Met-Glu-His-Phe-Pro-Gly-Pro
Brand Name N/A
Drug Class Peptide
CAS Number 80714-61-0
InChIKey AFEHBIGDWIGTEH-CXFOGXNKSA-N
Molecular Formula C37H51N9O10S
Molecular Weight 813.928 g/mol
Monoisotopic Mass 813.348 g/mol
Melting Point  N/A
Freezing Point N/A
Biological Half-Life N/A
Color white to tan
Solubility  H2O: >2 mg/mL
Storage Temperature  −20°C
Application Raw Semax powder is used for the treatment of brain hypoxia and ischemia

 


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COA

 photo COA 80714-61-0 Semax Phcoker_zpswotyn0kx.jpg

References & product citations

Top 7 Benefits of Semax You Should Know Before Use

 

References

Dolotov, O. V., Eremin, K. O., Andreeva, L. A., Novosadova, E. V., Raevskii, K. S., Myasoedov, N. F., & Grivennikov, I. A. (2015). Semax prevents the death of tyrosine hydroxylase-positive neurons in a mixed neuroglial cell culture derived from the embryonic rat mesencephalon in a model of 6-hydroxydopamine-induced neurotoxicity. Neurochemical Journal9(4), 295-298.

Koroleva, S. V., & Myasoedov, N. F. (2018). Semax as a Universal Drug for Therapy and Research. Biology Bulletin45(6), 589-600.

Manchenko, D. M., Glazova, N., Levitskaia, N. G., Andreeva, L. A., Kamenskiĭ, A. A., & Miasoedov, N. F. (2010). Nootropic and analgesic effects of Semax following different routes of administration. Rossiiskii fiziologicheskii zhurnal imeni IM Sechenova96(10), 1014-1023.

Malyshev, A. V., Razumkina, E. V., Dubynin, V. A., & Myasoedov, N. F. (2013, May). Semax corrects brain dysfunction caused by prenatal introduction of valproic acid. In Doklady Biological Sciences (Vol. 450, No. 1, p. 126). Springer Science & Business Media.